I have a 10 year old, male Pueblan Milk Snake who shed his skin just last week. Today, he's clouded up again, going immediately back into the "shed mode". As customary, he's slowed down considerably in his feeding during the winter months; and has no apparent injury. I know power feeding and injuries lessen the length of time between sheds, but not to this degree; and neither is the situation here.

Any ideas if this is something to be concerned about?

Thanks in advance,
Any ideas if this is something to be concerned about?

I would say, "yes, you should be concerned." As far as specific reasons WHY you should be concerned, I don't know, since I don't currently keep snakes and haven't had the rapid-shed problem. Emotion: sad
Have you done a Google search on the condition?
Thanks for your response, Chris; and yes I did the Google search, but found nothing relevant.
Thanks for your response, Chris; and yes I did the Google search, but found nothing relevant.

Are the back issues of Reptiles magazine archived/searchable online? You could try there, as I think the topic may have been covered in the past few years. If not, you could email their veterinarian and see if you get a response.
Lifted directly from one of Fr0glet's old posts:
From Biomedical and Surgical Aspects of Captive Reptile Husbandry ,vol 1, p176, Dr. Fredric Frye 1991 (thanks again Barbara for the awesome wedding gift!!!):

"Another form of dysecdysis observed in snakes in one in which the animal is almost constantly in some phase of molting. The interval between molts may be as brief as two weeks. Almost immediately after a successful molt, the snake's eyes again become opaque. The affected animal has to draw upon its body stores to complete the molt and synthesize a protein-rich replacement epidermis. This process is impaired because compounding the metabolic stress incurred during these almost continuous episodes of shedding is the fact that most snakes at this time are totally anorectic and refuse even preferred food items. Therefore, many of these animals are in negative nitrogen balance. (Doubt that's a problem with fat Sunny!!)

"The cases of this interesting disorder may be numerous and varied but, most frequently, the condition involves one or two thyroid dysfunctions: 1) primary hyperthyroidism or 2) mediation of a pituitary dysfunction that induces increased secretion of thyrotrophic hormone (TSH) and/or failure of the TSH inhibitory feedback loop to control the secretion of thyroxin. The effect in either situation is functional hyperthyroidism. Accurate diagnosis rests upon determination of thyroid function. Radioimmunoassay systems are extremely sensitive and provide reproducible results. Thyroid activity in an affected snake must be compared with that in a clinically normal snake of similar genus, sex, and size, and that has been maintained under similar captive environmental conditions. Obviously, this is not always practical or event possible. Maderson's elegant monograph published in 1985 goes into great detail on the subject of integumentary renewal.
"(... comparative treatments with lizards...) These results can be paradoxical but, clinically, most of the snakes with pathologically frequent shedding that the author has observed have improved after treatment with propylthiouracil at a dosage rate of 10 mg/kg p.o. once daily for at least 21-30 days by exhibiting a marked increase in the interval between molts. If methimazole is chosen as the antithyroid drug, its oral dosage is 1.0 to 1.25 mg/kg daily for 30 days. Both drugs inhibit the synthesis of thyroxine, but do not prevent the release of preformed thyroid hormone from the thyroid follicles. It is for this reason that either drug must be given for a sufficiently long period to expend any thyroxine-rich colloid that existed within the thyroid follicles prior to treatment."
There is much, much more on optional treatments

I was going to summarize it, but ctrl-C is so much quicker. Enjoy!